STANFORD SCIENTISTS CURE TYPE 1 DIABETES IN MICE WITH IMMUNE “REBOOT” TRANSPLANT

A breakthrough dual-transplant technique stops the autoimmune attack without insulin or immunosuppressants — offering hope for future human treatments.

Researchers at Stanford University, led by Dr. Seung K. Kim, have successfully cured type 1 diabetes in mice using a groundbreaking treatment that combines blood stem cell transplants with donor pancreatic islets — even when donor and recipient were immunologically incompatible.

The study, published in the Journal of Clinical Investigation, describes a procedure that “reboots” the immune system, stopping it from attacking insulin-producing beta cells. Remarkably, the treated mice required no chronic immunosuppressive drugs and no external insulin during six months of follow-up.


HOW THE TREATMENT WORKS

The therapy involves:

  1. Transplanting donor blood stem cells, which reset the immune system.
  2. Transplanting pancreatic islet cells, which restore natural insulin production.

This dual approach prevents both graft rejection and autoimmune destruction of beta cells — the two major obstacles to curing type 1 diabetes.

The results were extraordinary:

  • 19 out of 19 healthy mice had complete protection of donor islets.
  • 9 out of 9 diabetic mice were fully cured, maintaining normal glucose levels.

IMPLICATIONS FOR HUMANS

The breakthrough offers real hope for a future cure that:

  • Eliminates lifelong insulin injections
  • Removes the need for toxic immunosuppressants
  • Restores natural insulin regulation

Researchers believe the technique could also be adapted to treat other autoimmune diseases, including rheumatoid arthritis and lupus.

However, challenges remain — particularly the limited supply of donor islets from deceased donors. Future studies may explore lab-grown or stem-cell-derived alternatives.

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